Fescue Dystocia An owner adopted a 5 year old Tennessee Walking Horse mare that was part of a group of horses seized from a farm in West Virginia. Among this group were several stallions and a number of mares of breeding age. When the mare was evaluated she was checked for pregnancy and found to be in foal, but due to the nature of the history an exact breeding date/foaling date could not be established. The mares condition improved dramatically at her new home. She was given access to grass pasture while trying to limit the amount of access fescue. Even though she was on a very small amount of fescue it still affected her pregnancy. She showed no outward signs of impending parturition as fescue blocks the production of milk. Fescue toxicity causes premature placental separation, placentitis, prolonged gestation length, weak or dead foal, and agalactia (lack of milk production). The mare presented in labor, with no visible progress, on a weekday morning. On palpation both front feet of the foal were in the correct position, but the head was twisted backwards. Normal foaling position is a "diving" position with the front feet coming out first with the head between them. The foal was repositioned (much harder than it sounds!) and successfully delivered on the farm. The foal was dysmature, unable to stand on his own and had a weak suckle reflex (due to the fescue), a condition often referred to as a "dummy" foal. The mare had no colostrum (the first milk that provides antibodies for immune protection of the foal). The owners elected to take the mare and foal to a intensive care hospital due to the intensive care of hourly feedings, intravenous fluids and monitoring of a down foal . The foal took several days to stand on his own and required plasma to provide the antibodies needed due to the lack of colostrum. The mare was dehydrated from the ordeal and also retained her placenta due to the difficult birth and fescue toxicity (anything over 3 hours is considered retained). She was also treated to stimulate milk production. Both mare and foal recovered and are now home playing in their pasture and doing very well. Fescue toxicosis can be prevented by removing the mare from all fescue (including hay) before 300 days of gestation. If this is impossible the mare can be placed on a daily oral paste at 300 days of gestation. Dr. Lindberg with mare and foal at 4 weeks Hind Limb Laminitis Hind limb laminitis is usually the result of an injury to the other hind leg resulting in the horse bearing too much weight on the un-injured leg. In this case there was no apparent cause for the laminitis. Front limb laminitis is far more common, it has multiple causes including rich pasture, metabolic disease, grain overload. In mild cases, horses will be tender in both front feet. In a severe cases they will often be unwilling to move very far and will have their front feet out in front of their body in the classic "saw-horse" stance. Moldy Corn Poisoning History: A call was received from an owner of two miniature donkeys. The donkeys had a three day history of hind end weakness and being unable to rise. Both donkeys were bright, alert, responsive, and willing to eat and drink. Physical Exam: When we arrived at the farm one of the donkeys was recumbent (down) and had been that way overnight. No physical abnormalities were noted, yet the donkey was only able to rise and stand with assistance. The other donkey was standing and able to ambulate (walk) quite well. She was interested in eating and her physical exam was within normal limits, even though she was reported to need help rising. When more than one animal in a herd is affected with similar signs some type of toxin or poison is suspected. It was determined that both donkeys were eating corn that was manufactured for deer as part of their diet. Corn is often implicated as a carrier of Fumonisins, a mycotoxin which causes damage to the brain. Moldy Corn Poisoning: Moldy Corn Poisoning (Equine Leukoencephalomalacia or Fumonisin Toxicity) is a generally fatal, rapidly progressing neurologic disease of horses caused by ingestion of fumonisin. The toxin destroys a portion of the brain (cerebral white matter). Liver lesions can also occur. Fumonisins are environmental toxins produced by the molds Fusarium moniliforme (F. verticilloides), F. proliferatum, and other Fusarium species that grow on corn in the field or during storage. These mycotoxins have been found as common contaminants worldwide. The most prevalent mycotoxins in contaminated corn is FB1 which is believed to be the most toxic. The extent of contamination is affected by location the corn was grown, weather conditions, storage practices, and insect damage. Horses, along with rabbits, are most susceptible to poisoning from moldy corn. Ruminants (cows, sheep, deer, etc.) are able to ferment poor quality feedstuff in there rumen (large compartment of the stomach) before it is passed into the intestines for absorption, therefore are generally able to consume contaminated corn. Horses and rabbits lack this ability and must eat much higher quality feeds. Onset of clinical signs can can range from 2-9 weeks. This is dependent on concentration of toxin in the feed and amount consumed. After onset of signs death generally occurs in 12 hours to 1 week. Once an animal is unable to rise death generally occurs in 24-72 hours. Treatment: There is no specific treatment for moldy corn poisoning. Supportive care should be provided. Potent anti-inflammatory drugs may be beneficial in cases caught very early. Both of the donkeys were ultimately humanely euthanized due to progression of signs and poor prognosis. Navicular Bone Fracture Radiographs were taken to assess the boney structures located in the hoof. Theses structures include the coffin bone and navicular bone. Radiographs revealed that the navicular bone was fractured and there were three chips present. Surgical intervention is not an option for this type of fracture in this location. The treatment consist of special shoeing with a twelve degree pad applied to the foot and confinement. The wedge will be decreased by three degrees every 4 to 6 weeks until she is able to wear a flat shoe. The wedge pad decreases the pressure applied to navicular bone by the deep digital flexor tendon. At this time horse remains comfortable with a guarded prognosis for return to athletic work. December 2011 Update Follow up radiographs at 1 year show the area is healing, although there is some alteration to the surrounding tissue. There may be some residual lameness, but at this point in time the horse is sound in the field. |




